Güler, KadirTurhanoğlu, Ayşe Dicle2021-10-072021-10-072020GÜLER K,TURHANOĞLU A. D (2020). Does B12 deficiency lead to syringomyelia?. Turkish journal of physical medicine and rehabilitation, 66(3), 370 - 371. Doi: 10.5606/tftrd.2020.50402587-08232587-1250https://dx.doi.org/10.5606/tftrd.2020.5040https://hdl.handle.net/20.500.12483/3333A 20-year-old female was admitted to our clinic with the complaint of gait disturbance, which started three months ago and slowly progressed. Her medical history was non-specific. Her initial complaints began with numbness in her right foot and, then, under the left knee. Over time, activities such as sitting and standing up and climbing stairs became more difficult, and urinary incontinence started. Her physical examination revealed an ataxic gait pattern. Bilateral lower extremity strength was 4/5. Bilateral patellar reflexes were hyperactive. Bilateral Achilles reflexes were hypoactive. The plantar reflex was unresponsive at right and flexor at left. The superficial sensation was normal at lower extremities; however, there was a loss of deep sensation. The Romberg sign was positive. Laboratory test revealed that hemoglobin was 11 g/dL (reference: 13.6 to 17.2), mean corpuscular volume was 124 fL (reference: 80.4-95.9), serum vitamin B12 level was 80 pg/mL (reference: 190911), and liver and kidney function tests, electrolyte levels, blood glucose, glycosylated hemoglobin, C-reactive protein, and erythrocyte sedimentation rate were within normal limits. Nerve conduction studies showed that bilateral common peroneal, tibial, ulnar, and median nerve sensory and motor conduction were within normal limits.eninfo:eu-repo/semantics/openAccessArticle66337037110.5606/tftrd.2020.5040330890962-s2.0-85091210162Q2429408WOS:000569098300018Q4