Impact of chronic total occlusion in a non-infarct-related coronary artery on contrast-associated nephropathy in acute ST-elevation myocardial infarction

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dc.authoridCAKIR, BANU/0000-0001-6645-6527
dc.authoridYilmaz, Efe/0000-0003-2976-3063
dc.contributor.authorGok, Murat
dc.contributor.authorKurtul, Alparslan
dc.contributor.authorTaylan, Gokay
dc.contributor.authorOzturk, Cihan
dc.contributor.authorCakir, Burcu
dc.contributor.authorYilmaz, Efe
dc.contributor.authorAltay, Servet
dc.date.accessioned2024-09-18T20:13:19Z
dc.date.available2024-09-18T20:13:19Z
dc.date.issued2023
dc.departmentHatay Mustafa Kemal Üniversitesien_US
dc.description.abstractObjectives Co-existing chronic total occlusion (CTO) in a non-infarct-related artery (IRA) might serve as an important trigger of adverse outcomes in ST-segment elevation myocardial infarction (STEMI). Therefore, we planned to analyse the potential impact of non-IRA CTO on the evolution of contrast-associated nephropathy (CAN) in STEMI patients managed with primary percutaneous coronary intervention (P-PCI). Methods A total of 537 subjects with STEMI undergoing P-PCI during the first 12 h after the onset of their symptoms were enrolled in this retrospective study. The subjects were categorised based on the angiographic presence of non-IRA CTO. Moreover, the subjects were also divided into 2 groups based on their CAN status following P-PCI (CAN (+) and CAN (-)). Results Co-existing non-IRA CTO was demonstrated in 86 subjects (16%). During the hospitalisation period, we identified 81 (15.1%) subjects with CAN. Subjects with non-IRA CTO had a significantly higher incidence of CAN compared with those without (56 [12.4%] vs 25 [29.1%], respectively, p < 0.001). In a logistic regression analysis, an existing non-IRA CTO (odds ratio: 2.840, 95%CI: 1.451-5.558, p = 0.002), as well as age, haemoglobin, diabetes mellitus, creatinine, and white blood cell count, were independent of predictors of CAN. Conclusion In STEMI patients managed with P-PCI, a co-existing non-IRA CTO had an independent association with the evolution of CAN.en_US
dc.identifier.doi10.1080/00015385.2022.2085357
dc.identifier.endpage123en_US
dc.identifier.issn0001-5385
dc.identifier.issn1784-973X
dc.identifier.issue1en_US
dc.identifier.pmid35678246en_US
dc.identifier.scopus2-s2.0-85131700839en_US
dc.identifier.scopusqualityQ3en_US
dc.identifier.startpage118en_US
dc.identifier.urihttps://doi.org/10.1080/00015385.2022.2085357
dc.identifier.urihttps://hdl.handle.net/20.500.12483/9088
dc.identifier.volume78en_US
dc.identifier.wosWOS:000808503500001en_US
dc.identifier.wosqualityQ4en_US
dc.indekslendigikaynakWeb of Scienceen_US
dc.indekslendigikaynakScopusen_US
dc.indekslendigikaynakPubMeden_US
dc.language.isoenen_US
dc.publisherTaylor & Francis Ltden_US
dc.relation.ispartofActa Cardiologicaen_US
dc.relation.publicationcategoryMakale - Uluslararası Hakemli Dergi - Kurum Öğretim Elemanıen_US
dc.rightsinfo:eu-repo/semantics/closedAccessen_US
dc.subjectContrast-associated nephropathyen_US
dc.subjectchronic total occlusionen_US
dc.subjectnon-infarct-related arteryen_US
dc.subjectprimary percutaneous coronary interventionen_US
dc.subjectST-segment elevation myocardial infarctionen_US
dc.titleImpact of chronic total occlusion in a non-infarct-related coronary artery on contrast-associated nephropathy in acute ST-elevation myocardial infarctionen_US
dc.typeArticleen_US

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