Basal Septal Hypertrophy as the Early Imaging Biomarker for Adaptive Phase of Remodeling Prior to Heart Failure

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dc.authoridKucukler, Nagehan/0000-0002-9867-266X
dc.authoridakkus, oguz/0000-0001-7988-7027
dc.contributor.authorYalcin, Fatih
dc.contributor.authorYalcin, Hulya
dc.contributor.authorKucukler, Nagehan
dc.contributor.authorArslan, Serbay
dc.contributor.authorAkkus, Oguz
dc.contributor.authorKurtul, Alparslan
dc.contributor.authorAbraham, Maria Roselle
dc.date.accessioned2024-09-18T20:02:54Z
dc.date.available2024-09-18T20:02:54Z
dc.date.issued2022
dc.departmentHatay Mustafa Kemal Üniversitesien_US
dc.description.abstractHypertension plays a dominant role in the development of left ventricular (LV) remodeling and heart failure, in addition to being the main risk factor for coronary artery disease. In this review, we focus on the focal geometric and functional tissue aspects of the LV septal base, since basal septal hypertrophy (BSH), as the early imaging biomarker of LV remodeling due to hypertensive heart disease, is detected in cross-sectional clinic studies. In addition, the validation of BSH by animal studies using third generation microimaging and relevant clinical observations are also discussed in the report. Finally, an evaluation of both human and animal quantitative imaging studies and the importance of combined cardiac imaging methods and stress-induction in the separation of adaptive and maladaptive phases of the LV remodeling are pointed out. As a result, BSH, as the early imaging biomarker and quantitative follow-up of functional analysis in hypertension, could possibly contribute to early treatment in a timely fashion in the prevention of hypertensive disease progression to heart failure. A variety of stress stimuli in etiopathogenesis and the difficulty of diagnosing pure hemodynamic overload mediated BSH lead to an absence of the certain prevalence of this particular finding in the population.en_US
dc.identifier.doi10.3390/jcm11010075
dc.identifier.issn2077-0383
dc.identifier.issue1en_US
dc.identifier.pmid35011816en_US
dc.identifier.scopus2-s2.0-85121659979en_US
dc.identifier.scopusqualityQ1en_US
dc.identifier.urihttps://doi.org/10.3390/jcm11010075
dc.identifier.urihttps://hdl.handle.net/20.500.12483/8082
dc.identifier.volume11en_US
dc.identifier.wosWOS:000751033100001en_US
dc.identifier.wosqualityQ2en_US
dc.indekslendigikaynakWeb of Scienceen_US
dc.indekslendigikaynakScopusen_US
dc.indekslendigikaynakPubMeden_US
dc.language.isoenen_US
dc.publisherMdpien_US
dc.relation.ispartofJournal of Clinical Medicineen_US
dc.relation.publicationcategoryMakale - Uluslararası Hakemli Dergi - Kurum Öğretim Elemanıen_US
dc.rightsinfo:eu-repo/semantics/openAccessen_US
dc.subjectbasal septal hypertrophyen_US
dc.subjectearly imaging biomarkeren_US
dc.subjectheart failureen_US
dc.subjecthypertension and stressed heart morphologyen_US
dc.titleBasal Septal Hypertrophy as the Early Imaging Biomarker for Adaptive Phase of Remodeling Prior to Heart Failureen_US
dc.typeArticleen_US

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