Left renal atrophy

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dc.contributor.authorDavran, Ramazan
dc.contributor.authorHelvaci, Mehmet Rami
dc.contributor.authorDavarci, Mursel
dc.date.accessioned2024-09-18T20:29:32Z
dc.date.available2024-09-18T20:29:32Z
dc.date.issued2014
dc.departmentHatay Mustafa Kemal Üniversitesien_US
dc.description.abstractBackground: We tried to understand whether or not there is a higher risk of left renal atrophy in human being. Methods: All patients applying to the Hematology Service with any underlying complaint were studied. Results: The study included 2,417 cases (1,248 females). The mean ages were 47.3 versus 50.7 years in females and males, respectively (p<0.000). There were 33 cases (1.3%) with the left renal atrophy against five cases (0.2%) with the right (p<0.001). The left renal atrophy cases have splenomegaly (SM) in 51.5%, thalassemia minors (TMs) in 30.3%, sickle cell diseases (SCDs) in 27.2%, myeloproliferative disorders in 18.1%, chronic lymphocytic leukemia in 6.0%, cirrhosis in 6.0%, solid organ malignancies in 6.0%, chronic obstructive pulmonary disease in 3.0%, multiple myeloma in 3.0%, and Waldenstrom's macroglobulinemia in 3.0%. Similarly, the right renal atrophy cases have SM in 20.0%, TMs in 40.0%, and SCDs in 20.0%. Conclusion: Left renal atrophy may be significantly higher than the right side in human being. Aortic pressure induced flow disorders in the left renal vein, structural anomalies of the left renal vein, and possibly the higher arterial pressure of the left kidney due to the shorter distance to the heart as an underlying cause of atherosclerosis may be some of the possible causes. Due to the stronger arterial wall protecting itself from compression and high prevalences of SM and left varicocele in population, SM induced flow disorders of the left renal vein may be the most common cause.en_US
dc.identifier.endpage1606en_US
dc.identifier.issn1940-5901
dc.identifier.issue6en_US
dc.identifier.pmid25035786en_US
dc.identifier.scopus2-s2.0-84904042203en_US
dc.identifier.scopusqualityN/Aen_US
dc.identifier.startpage1603en_US
dc.identifier.urihttps://hdl.handle.net/20.500.12483/10915
dc.identifier.volume7en_US
dc.identifier.wosWOS:000341255900016en_US
dc.identifier.wosqualityQ4en_US
dc.indekslendigikaynakWeb of Scienceen_US
dc.indekslendigikaynakScopusen_US
dc.indekslendigikaynakPubMeden_US
dc.language.isoenen_US
dc.publisherE-Century Publishing Corpen_US
dc.relation.ispartofInternational Journal of Clinical and Experimental Medicineen_US
dc.relation.publicationcategoryMakale - Uluslararası Hakemli Dergi - Kurum Öğretim Elemanıen_US
dc.rightsinfo:eu-repo/semantics/closedAccessen_US
dc.subjectLeft renal atrophyen_US
dc.subjectsplenomegalyen_US
dc.subjectleft renal veinen_US
dc.subjectatherosclerosisen_US
dc.titleLeft renal atrophyen_US
dc.typeArticleen_US

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