Does B12 deficiency lead to syringomyelia?

dc.authorid0000-0001-9465-6665en_US
dc.authorid0000-0001-6273-9589en_US
dc.contributor.authorGüler, Kadir
dc.contributor.authorTurhanoğlu, Ayşe Dicle
dc.date.accessioned2021-10-07T12:33:23Z
dc.date.available2021-10-07T12:33:23Z
dc.date.issued2020en_US
dc.departmentTayfur Ata Sökmen Tıp Fakültesien_US
dc.description.abstractA 20-year-old female was admitted to our clinic with the complaint of gait disturbance, which started three months ago and slowly progressed. Her medical history was non-specific. Her initial complaints began with numbness in her right foot and, then, under the left knee. Over time, activities such as sitting and standing up and climbing stairs became more difficult, and urinary incontinence started. Her physical examination revealed an ataxic gait pattern. Bilateral lower extremity strength was 4/5. Bilateral patellar reflexes were hyperactive. Bilateral Achilles reflexes were hypoactive. The plantar reflex was unresponsive at right and flexor at left. The superficial sensation was normal at lower extremities; however, there was a loss of deep sensation. The Romberg sign was positive. Laboratory test revealed that hemoglobin was 11 g/dL (reference: 13.6 to 17.2), mean corpuscular volume was 124 fL (reference: 80.4-95.9), serum vitamin B12 level was 80 pg/mL (reference: 190911), and liver and kidney function tests, electrolyte levels, blood glucose, glycosylated hemoglobin, C-reactive protein, and erythrocyte sedimentation rate were within normal limits. Nerve conduction studies showed that bilateral common peroneal, tibial, ulnar, and median nerve sensory and motor conduction were within normal limits.en_US
dc.identifier.citationGÜLER K,TURHANOĞLU A. D (2020). Does B12 deficiency lead to syringomyelia?. Turkish journal of physical medicine and rehabilitation, 66(3), 370 - 371. Doi: 10.5606/tftrd.2020.5040en_US
dc.identifier.doi10.5606/tftrd.2020.5040en_US
dc.identifier.endpage371en_US
dc.identifier.issn2587-0823
dc.identifier.issn2587-1250
dc.identifier.issue3en_US
dc.identifier.pmid33089096en_US
dc.identifier.scopus2-s2.0-85091210162en_US
dc.identifier.scopusqualityQ2en_US
dc.identifier.startpage370en_US
dc.identifier.trdizinid429408en_US
dc.identifier.urihttps://dx.doi.org/10.5606/tftrd.2020.5040
dc.identifier.urihttps://hdl.handle.net/20.500.12483/3333
dc.identifier.volume66en_US
dc.identifier.wosWOS:000569098300018en_US
dc.identifier.wosqualityQ4en_US
dc.indekslendigikaynakWeb of Scienceen_US
dc.indekslendigikaynakScopusen_US
dc.indekslendigikaynakPubMeden_US
dc.indekslendigikaynakTR-Dizinen_US
dc.language.isoenen_US
dc.publisherTurkish Society of Physical Medicine and Rehabilitationen_US
dc.relation.ispartofTurkish Journal of Physical Medicine and Rehabilitationen_US
dc.relation.publicationcategoryMakale - Uluslararası Hakemli Dergi - Kurum Öğretim Elemanıen_US
dc.rightsinfo:eu-repo/semantics/openAccessen_US
dc.titleDoes B12 deficiency lead to syringomyelia?en_US
dc.typeArticleen_US

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